Letter to the editorAssociations between prenatal pesticide exposure and cough, wheeze, and IgE in early childhood
References (14)
- et al.
Anti-cockroach and anti-mouse IgE are associated with early wheeze and atopy in an inner city birth cohort
J Allergy Clin Immunol
(2008) - et al.
Mechanisms of pyrethroid neurotoxicity: implications for cumulative risk assessment
Toxicology
(2002) - et al.
Pesticides associated with wheeze among commercial pesticide applicators in the Agricultural Health Study
Am J Epidemiol
(2006) - et al.
Pesticides and atopic and nonatopic asthma among farm women in the Agricultural Health Study
Am J Respir Crit Care Med
(2008) - et al.
Early-life environmental risk factors for asthma: findings from the Children's Health Study
Environ Health Perspect
(2004) - et al.
Early environmental exposures and intracellular Th1/Th2 cytokine profiles in 24-month-old children living in an agricultural area
Environ Health Perspect
(2006) - et al.
Plagued by pesticides: an analysis of New York State's 1997 pesticide use and sales data
(1998)
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Exposure to biocides and its association with atopic dermatitis among children and adolescents: A population-based cross-sectional study in South Korea
2024, Ecotoxicology and Environmental SafetyAge-related differences in IgE between childhood and adulthood allergic asthma: Analysis of NHANES 2005–2006
2023, World Allergy Organization JournalMother/child organophosphate and pyrethroid distributions
2020, Environment InternationalCitation Excerpt :Postnatal exposure to these compounds has also been reported deleterious for neurodevelopment, which include mainly deficits in working memory, low attention or motor speed (Ruckard et al., 2004; Rohlman et al., 2005). Exposure to PYR residues has been significantly associated with increases of autism spectrum disorder (von Ehrenstein et al., 2019) and respiratory outcomes (Reardon et al., 2009). A meta-analysis study on the daily intake and risk assessment of OPs in farmers, general population, children and pregnant women observed differential exposures depending on occupation and age (Katsikantami et al., 2019).
Supported by grants National Institutes of Health P01 ES09600, ES008977, P30 ES009089, Environmental Protection Agency RD 83214101, and Environmental Protection Agency R827027.
Disclosure of potential conflict of interest: R. L. Miller receives grant support from the National Institutes of Health, the Environmental Protection Agency, and the Sandeer Program for Asthma Research and is a volunteer member of the American Thoracic Society. M. S. Perzanowski receives honoraria from Indoor Biotechnologies and receives grant support from the National Institutes of Health. R. M. Whyatt receives grant support from the National Institutes of Health and the Environmental Protection Agency. G. L. Chew receives grant support from the National Institute of Environmental Health Sciences. The rest of the authors have declared that they have no conflict of interest.