Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology
Edema in the upper airway in patients with obstructive sleep apnea syndrome
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Cited by (40)
Renal disorders and sleep
2023, Encyclopedia of Sleep and Circadian Rhythms: Volume 1-6, Second EditionSleep and coronary artery disease risk
2014, Indian Journal of Medical SpecialitiesCitation Excerpt :Serotonin is thought to be an important mediator of upper airway dilating muscle activity and is reduced during sleep in patients with OSAHS. Magnetic resonance imaging32 and histological studies33 have demonstrated that patients with OSAHS develop upper airway oedema secondary to the mechanical trauma associated with snoring and recurrent upper airway obstruction. In short, sleep onset is associated with decreased upper airway muscle activity, which further results in upper airway collapse and hypoventilation.
Contrasting effects of lower body positive pressure on upper airways resistance and partial pressure of carbon dioxide in men with heart failure and obstructive or central sleep apnea
2013, Journal of the American College of CardiologyCitation Excerpt :Compared with age- and weight-matched controls, OSA patients have higher pharyngeal resistance, and greater compliance and collapsibility during both wakefulness and sleep (38–41). Pharyngeal narrowing and increased Rph can be due to an increase in surrounding soft tissue as a result of muscular hypertrophy, inflammation, and fat or fluid accumulation in the peripharyngeal tissues (4,36). The latter may be amenable to manipulation.
Patent foramen ovale-obstructive sleep apnea relationships: Pro and cons
2012, Cardiovascular Revascularization MedicineCitation Excerpt :Obstructive apnea may be considered as a succession of Valsalva and Mueller manoeuvres as the patient expires and inspires against an occluded airway. Indeed many patients with OSASH have also increased upper airway resistance because of different reasons including interstitial oedema with a mild inflammatory reaction [16] and diminution of pharyngeal diameter [17]. The role of increased airway resistance in generating RLS through PFO is also supported by the increased prevalence of PFO in patients with chronic obstructive pulmonary disease [18].
Cephalometric and three-dimensional assessment of superior posterior airway space after maxillomandibular advancement
2012, International Journal of Oral and Maxillofacial Surgery