Characterization of contractile prostanoid receptors on human airway smooth muscle
References (26)
- et al.
Potentiation of contraction of rabbit airway smooth muscle by some cyclooxygenase products
Prostaglandins
(1988) - et al.
The calcium dependence of histamine, carbachol and potassium chloride-induced contraction in human airways in vitro
European J. Pharmacol.
(1986) - et al.
A comparison of the contractile activity of PGD2 and PGF2α on human isolated bronchus
Prostaglandins
(1986) - et al.
Specific receptors for prostaglandins in airways
Prostaglandins
(1980) - et al.
Studies on the characterization of prostanoid receptors: a proposed classification
Prostaglandins
(1982) - et al.
Extracellular calcium and human isolated airway muscle: ionophore A23187 induced contraction
Respir, Physiol.
(1988) - et al.
Characterization of receptors mediating the contractile effects of prostanoids in guinea-pig and human airways
European J. Pharmacol.
(1988) - et al.
A role for inflammatory mediators in airway hyperresponsiveness
- et al.
Formyl peptide-induced contraction of human airways in vitro
J. Appl. Physiol.
(1986) - et al.
Relationship between smooth muscle volume and contractile response in airway tissue. Isometri vs isotonic measurement
J. Pharmacol. Exp. Ther.
(1988)
Some quantitative uses of drug antagonists
Br. J. Pharmacol.
The interaction between indomethacin and contractile agents on human isolated airway muscle
Br. J. Pharmacol.
Receptor mechanisms for 5-hydroxytryptamine in rabbit arteries
Br. J. Pharmacol.
Cited by (58)
Defining the contractile prostanoid component in hyperosmolar-induced bronchoconstriction in human small airways
2023, Prostaglandins and Other Lipid MediatorsSex steroids effects on guinea pig airway smooth muscle tone and intracellular Ca<sup>2+</sup> basal levels
2017, Molecular and Cellular EndocrinologyCitation Excerpt :It is well known that among all prostanoids, the main three that can be produced in airway smooth muscle and have an effect on this tissue are: thromboxane A2 (TXA2), prostaglandin F2α (PGF2α) and PGE2. Regarding TXA2 and PGF2α, it was reported (Armour et al., 1989) that they induce airway smooth muscle contraction through the activation of a TP receptor. Later on, it was stated that these prostanoids also augmented intracellular Ca2+ concentrations (Shiraki et al., 2009; Takata et al., 1999).
Prostaglandin E<inf>2</inf> inhibits mast cell–dependent bronchoconstriction in human small airways through the E prostanoid subtype 2 receptor
2015, Journal of Allergy and Clinical ImmunologyLipid Mediators and Lung Function
2015, Comparative Biology of the Normal Lung: Second EditionModulation of antigen-induced responses by serotonin and prostaglandin E<inf>2</inf> via EP<inf>1</inf> and EP<inf>4</inf> receptors in the peripheral rat lung
2013, European Journal of PharmacologyCitation Excerpt :TXA2 may also cause airway hyperresponsiveness (Held and Uhlig, 2000) and contributes to cytokine-induced bronchoconstriction (Martin et al., 2001). PGD2 is a pro-inflammatory mediator of allergic asthma (Matsuoka et al., 2000), a marker of mast cell activation (Dahlen and Kumlin, 2004) and induces airway and vascular smooth muscle contractions via the TP receptor (Armour et al., 1989; Beasley et al., 1989; Johnston et al., 1995; Larsson et al., 2011; McKenniff et al., 1991). PGE2 is implicated to have a beneficial role in the lung (Pavord and Tattersfield, 1995; Vancheri et al., 2004), since this prostanoid may maintain airway tone (Tilley et al., 2003) and attenuate allergic airway responses (Hartney et al., 2006; Martin et al., 2002).