Effect of hypoxemia on sodium and water excretion in chronic obstructive lung disease☆
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Cited by (43)
The prevalence of the syndrome of inappropriate antidiuretic hormone secretion (SIADH) in brucellosis patients: Systematic review and meta-analysis
2022, Annals of Medicine and SurgeryCitation Excerpt :Direct involvement of the central nervous system can lead to ectopic ADH secretion and SIADH [17]. Decreased venous return, hypoxemia, and hypercapnia that occasionally accompany lung infections are proposed to induce a non-osmotic ADH release [18–22]. Regulation of serum sodium levels assuming at newer lower target (e.g., 125–135 mmol/L), also known as reset osmostat, was described in malaria and tuberculosis cases [23,24].
Pulmonary Hypertension Due to Lung Disease
2015, Murray and Nadel's Textbook of Respiratory Medicine: Volume 1,2, Sixth EditionHyponatremia in patients with infectious diseases
2011, Journal of InfectionCitation Excerpt :In accordance with this hypothesis, low urine sodium has been reported in this clinical context.40 Moreover, hypoxemia and hypercapnia (usually observed in respiratory infections) alone or in combination can stimulate non-osmotic release of ADH.41,42 Reset osmostat, a variant of SIADH in which the serum sodium concentration is normally regulated (and is therefore stable) at a lower level (typically between 125 and 135 mmol/L), has been reported in infectious disease, such as tuberculosis and malaria.43,44
Protocolo terapéutico del cor pulmonale
2009, MedicineNeural and humoral control of the airways
2009, Asthma and COPD
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This work was supported in part by Grant HL-27294 from the United States Public Health Service.