@article {Recalde-Zamacona1116, author = {Borja Recalde-Zamacona and Laura Garc{\'\i}a-Tobar and Alan Argueta and Laura {\'A}lvarez and Carlos Eduardo De Andrea and Mirian Fern{\'a}ndez Alonso and Ana Ezponda and Francisco Carmona Torre and Carlota Jord{\'a}n Iborra and Jorge Augusto Quiroga and Jose Luis Del Pozo and Javier J Zulueta and Gema Echarri and Manuel F Landecho and Maria Dolores Lozano}, title = {Histopathological findings in fatal COVID-19 severe acute respiratory syndrome: preliminary experience from a series of 10 Spanish patients}, volume = {75}, number = {12}, pages = {1116--1118}, year = {2020}, doi = {10.1136/thoraxjnl-2020-215577}, publisher = {BMJ Publishing Group Ltd}, abstract = {In December 2019, an outbreak of severe acute respiratory syndrome associated to SARS-CoV2 was reported in Wuhan, China. To date, little is known on histopathological findings in patients infected with the new SARS-CoV2. Lung histopathology shows features of acute and organising diffuse alveolar damage. Subtle cellular inflammatory infiltrate has been found in line with the cytokine storm theory. Medium-size vessel thrombi were frequent, but capillary thrombi were not present. Despite the elevation of biochemical markers of cardiac injury, little histopathological damage could be confirmed. Viral RNA from paraffin sections was detected at least in one organ in 90\% patients.}, issn = {0040-6376}, URL = {https://thorax.bmj.com/content/75/12/1116}, eprint = {https://thorax.bmj.com/content/75/12/1116.full.pdf}, journal = {Thorax} }