Respiratory syncytial virus (RSV) and ΔSHRSV mediated mechanism of inflammasome activation. Lung epithelial cells were either infected with RSV or ΔSHRSV. Cell supernatant was collected and tested for IL1β (A), IL6 (B) and IFNβ (C) secretion. Cells extracts from infected lung epithelial cells were analysed for the presence of pro-IL1β (D) and Caspase p10 (E) by western blotting. The data presented are the mean±SD of three independent experiments.