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Correspondence
GWAS in lung disease
  1. Robert P Young1,2,
  2. Raewyn J Hopkins1,2
  1. 1School of Biological Sciences, University of Auckland, Auckland, New Zealand
  2. 2School of Health Sciences, University of Auckland, Auckland, New Zealand
  1. Correspondence to Dr Robert P Young, PO Box 26161, Epsom 1344, Auckland, New Zealand; roberty{at}adhb.govt.nz

https://doi.org/10.1136/thoraxjnl-2011-201034

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    We read with interest the recent article ‘Genome-wide association studies in lung disease’ by Artigas et al.1 While we agree that a greater understanding of the biological pathways underlying disease development and progression (susceptibility) will be a major outcome from these genetic epidemiological studies, we suggest other benefits may also stem from this research.

    The genetics of chronic obstructive pulmonary disease (COPD) and lung cancer represent unique models for the genetics of lung disease because they result in the main from a ‘single’ …

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    Footnotes

    • Funding RPY, and the funding of his research, has been supported by grants from the University of Auckland, Health Research Council of New Zealand and Synergenz BioSciences Ltd.

    • Competing interests None.

    • Provenance and peer review Not commissioned; internally peer reviewed.