T6 MET targeted therapy in Lung adenocarcinoma: Does ‘Resistant’ EGFR make a MET-responsive dimer? | Thorax

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Article info

Prize symposium

BTS/BLF/BALR Early Career Investigators Symposium

T6 MET targeted therapy in Lung adenocarcinoma: Does ‘Resistant’ EGFR make a MET-responsive dimer?

Free

Authors

RW Lee 1Division of Asthma, Allergy and Lung Biology, Guy’s Hospital, King’s Health Partners, London, UK PubMed articles Google scholar articles
E Ortiz-Zapater 1Division of Asthma, Allergy and Lung Biology, Guy’s Hospital, King’s Health Partners, London, UK PubMed articles Google scholar articles
G Weitsman 2Richard Dimbleby Department of Cancer Research, Randall Division of Cell and Molecular Biophysics, King’s College London, London, UK PubMed articles Google scholar articles
G Fruhwirth 3Department of Imaging Chemistry and Biology Division of Imaging Sciences and Biomedical Engineering School of Medicine, King’s College London, London, UK PubMed articles Google scholar articles
W Owen 1Division of Asthma, Allergy and Lung Biology, Guy’s Hospital, King’s Health Partners, London, UK PubMed articles Google scholar articles
T Ng 2Richard Dimbleby Department of Cancer Research, Randall Division of Cell and Molecular Biophysics, King’s College London, London, UK PubMed articles Google scholar articles
G Santis 1Division of Asthma, Allergy and Lung Biology, Guy’s Hospital, King’s Health Partners, London, UK PubMed articles Google scholar articles

Citation

Lee R, Ortiz-Zapater E, Weitsman G, et al

T6 MET targeted therapy in Lung adenocarcinoma: Does ‘Resistant’ EGFR make a MET-responsive dimer?

Thorax 2015;70:A3-A5.

Publication history

First published November 12, 2015.

Online issue publication

September 01, 2022

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